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By: alok, On Thursday, August 16th, 2018 In Uncategorized. No Comments

A few patients have developed parkinsonian crises, manifested as confusion, marked increase in rigidity, urinary retention, and bulbar palsy, within 3 days after abrupt discontinuance of the drug, even in the absence of apparent clinical improvement with amantadine therapy.

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An antivial that is usd in th pophylactic symptomatic tatmnt inlunza A. It is alsusd as an antipakinsonian agnt, ttat xtapyamidal actions, and posthptic nualgia. Th mchanisms its cts in movmnt disods a not wll undstood but pobably lct an incas in synthsis and las dopamin, with phaps som inhibition dopamin uptak. [PubChm]

Th chmopophylaxis, pophylaxis, and tatmnt signs and symptoms inction causd by vaious stains inlunza A vius. Alsth tatmnt pakinsonism and dug-inducd xtapyamidal actions.

Us this mdication gulaly tgt th most bnit om it. Thlp you mmb, tak it at th sam tim ach day.

Amantadin is an antivial dug which alsacts as an antipakinson agnt, which it is usually combind with L-DOPA whn L-DOPA sponss dclin (pobably du ttolanc). It is a divat adamantan, lik a simila dug imantadin. Th mchanism action amantadin in th tatmnt Pakinson’s disas and dug-inducd xtapyamidal actions is not known. It has bn shown tcaus an incas in dopamin las in th animal bain, and dos not possss anticholingic activity.

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